Cardiac amyloidosis: a review

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dc.contributor.author Baska, Aleksandra
dc.contributor.author Kazik, Jakub
dc.contributor.author Bojczuk, Daria
dc.contributor.author Racinowski, Mariusz
dc.contributor.author Leis, Kamil
dc.contributor.author Mazur, Ewelina
dc.contributor.author Kałużny, Krystian
dc.contributor.author Gałązka, Przemysław
dc.date.accessioned 2019-03-19T12:11:20Z
dc.date.available 2019-03-19T12:11:20Z
dc.date.issued 2019-03-03
dc.identifier.citation Journal of Education, Health and Sport, No. 3, Vol. 9, pp. 333-338.
dc.identifier.issn 2391-8306
dc.identifier.other doi:10.5281/zenodo.2597569
dc.identifier.uri http://repozytorium.umk.pl/handle/item/5816
dc.description.abstract Cardiac amyloidosis is a type of amyloidosis in which one of the affected organs is the heart. The disease is characterized by the formation of protein aggregates between the cells of the organ, namely amyloid, which disables its function. The following types of amyloidosis can be distinguished: systemic senile amyloidosis (wild-type ATTR), light chain amyloidosis (AL) and hereditary transthyretin-related amyloidosis. The symptoms include, e.g., systolic dysfunction or arrhythmia. The treatment is focused on the therapy with melphalan and, additionally, stem cells transplant and chemotherapy with dexamethasone or cyclophosphamide. In the advanced stage of the disease, a heart transplant is necessary. The diagnosis is made on the basis of laboratory testing, electrocardiogram changes, and echocardiography.
dc.language.iso eng
dc.rights Attribution-NonCommercial-ShareAlike 4.0 International
dc.rights info:eu-repo/semantics/openAccess
dc.rights.uri http://creativecommons.org/licenses/by-nc-sa/4.0/
dc.subject cardiac amyloidosis
dc.subject amyloid
dc.subject heart failure
dc.title Cardiac amyloidosis: a review
dc.type info:eu-repo/semantics/article

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